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We offer our services on Atresia, Dystocia and C's, herniorrhaphy (hernia), prolapse or abscess pus, mastitis, dermatitis, tetanus infection, poultry related cases and public health services.

Whipworms in dogsWhipworms are an intestinal parasite that occurs in dogs worldwide and get their name from the whip-lik...
27/06/2025

Whipworms in dogs

Whipworms are an intestinal parasite that occurs in dogs worldwide and get their name from the whip-like shape of the adult worms. Dogs get by whipworms after ingesting an egg that passes through the stool of an infected dog left in the soil or other contaminated environments. While some dogs may have no clinical signs of infection, others may experience intermittent soft stool or diarrhea with blood or mucus. Whipworm eggs are extremely hardy in the environment, so prevention with monthly parasite prevention is critical.

Cause

A nematode parasite, Trichuris vulpis causes whipworm infections in the large intestine (colon and cecum) in dogs. When an infected dog passes a whipworm egg in their stool, the egg is not infective to another dog (i.e., larvated) until 9-21 days in the environment. The eggs are quite hardy in the environment and can survive in the soil for years. Dogs become infected by whipworms after ingesting a larvated egg from a contaminated environment. The larvae enter the small intestine for a few days before moving to the colon and cecum to mature into adult whipworms. The adult worms take quite long before they start producing eggs (74-90 days), and then each female produces over 2,000 a day.

Clinical signs

Many dogs with whipworms may show no signs of infection. The most common clinical signs of whipworms may include any of the following:

Blood or mucus in f***s
Straining to defecate
Weight loss
Anemia, lethargy, pale mucus membranes
Soft stools or diarrhea
Dehydration
Decreased appetite
Severe whipworm infections may sometimes cause vomiting, weakness, decreased heart rate, muscle tremors and abdominal pain. Rarely, heavy whipworm infections can also cause a serious intestinal condition called intussusception or even death.

Diagnosis

Whipworms are diagnosed by bringing a f***l sample from your dog to your veterinarian to check for parasite eggs under a microscope (f***l flotation with centrifugation). Sometimes this test may not detect eggs despite the presence of a dogโ€™s clinical signs of infection due to the long time it takes for adult worms to produce eggs or intermittent shedding of eggs in the f***s. Repeat serial samples may be needed, or an ELISA test (Enzyme-Linked Immunosorbent Assay) can be used to detect whipworm antigens to confirm a diagnosis if whipworms are suspected but no eggs were detected.

If more significant signs of illness are present, your veterinarian may recommend additional testing such as bloodwork or abdominal imaging.

Treatment

Several anthelmintic medications, such as Panacur (fenbendazole) or Drontal Plus (febantel/pyrantel pamoate/praziquantel), are approved for treating whipworms in dogs. These treatments target adult worms only. Because the lifecycle of whipworms is quite long, this treatment must be repeated monthly for three months to treat the newly emerging adults as they mature.

Alternatively, after initial treatment, many monthly heartworm preventatives containing milbemycin or moxidectin (e.g., Interceptor, Sentinel, Trifexis, Advantage Multi) are approved for the monthly treatment and prevention of whipworms.

For more serious whipworm infections, fluid therapy or nutritional support may be needed.

Outcome

Most dogs with whipworms respond well to treatment and environmental control. Your veterinarian may recommend periodic f***l exams to monitor for infections. Persistent infection or reinfection is likely due to re-exposure to a contaminated environment.

Prevention

Prompt f***s removal to prevent contaminating the environment with whipworm eggs
Keep dogs on monthly parasite preventives to help prevent infections
Use caution when bringing pets to areas where other dogs frequent.

AMKOVET CONSULT.

Tetanus in livestock...Tetanus is a severe, often fatal, neuroparalytic disease of animals caused by the potent neurotox...
26/06/2025

Tetanus in livestock...

Tetanus is a severe, often fatal, neuroparalytic disease of animals caused by the potent neurotoxins produced by Clostridium tetani. Its insidious onset and characteristic clinical manifestations make it a significant concern in veterinary medicine, particularly in susceptible species like equines.

Aetiology and epidemiology
The causative agent, Clostridium tetani, is a ubiquitous, Gram-positive, anaerobic, spore-forming bacterium. It is widely distributed in the environment, particularly prevalent in cultivated soils where it can persist for long periods as resistant spores. Importantly, Clostridium tetani is also a normal flora inhabitant in the gastrointestinal tract (GIT) of equines, which can serve as a constant source of environmental contamination through their f***s.

Infection typically occurs when these spores are introduced into tissues through wounds. The critical factor for spore germination and subsequent toxin production is the creation of a suitable anaerobic environment within the wound. Deep, penetrating wounds, contaminated wounds (especially with soil or foreign bodies), puncture wounds (e.g., from nails, splinters), surgical incisions, or even umbilical infections in neonates can provide these anoxic conditions, allowing the dormant spores to revert to their vegetative, toxin-producing forms.

Pathogenesis
Following inoculation, the Clostridium tetani bacteria will localize within the necrotic tissues at the site of the injury and begin to multiply. As the bacteria multiply, they produce and release potent neurotoxins, primarily tetanospasmin. These toxins do not typically cause widespread systemic infection; rather, their localized production and subsequent action on the nervous system drive the disease.

The released neurotoxins will be absorbed into motor nerves around the infected area. Once absorbed, the toxin undergoes retrograde axonal transport, meaning it travels up the nerve tract to the spinal cord, and from there, it can ascend to the brain. Tetanospasmin specifically targets inhibitory interneurons in the spinal cord and brainstem. Within the brain, the toxins exert their primary effect by binding to presynaptic terminals and inhibiting the release of inhibitory neurotransmitters, such as glycine and gamma-aminobutyric acid (GABA). This blockade of inhibitory signals leads to the continuous, uncontrolled excitation of motor neurons, resulting in spasmodic tonic contractions of all the voluntary muscles. This sustained muscle contraction leads to rigidity, hypertonicity, and spasms, severely interfering with feeding, rest, [and] breathing.

Clinical signs: The progression of rigidity

The clinical signs of tetanus are a direct reflection of the neurotoxin's effects on the motor nervous system. Symptoms typically develop within days to weeks post-exposure, depending on the dose of toxin and the location of the wound.

The symptoms begin with spasms of head muscles, which is often one of the earliest and most recognizable signs. This makes it difficult to prehend or masticate food, leading to the characteristic "Locked Jaw" (or trismus). In horses and donkeys, highly susceptible species, the ears will become erect, the tail becomes stiff and extended (flagged), and the anterior (or hindlimb) muscles will become dilated (rigid or tense). A distinctive sign is the 3rd eyelid will prolapse across the eye, often visible even when the animal is calm, and becomes more pronounced with stimulation.

As the disease progresses, the generalized rigidity affects locomotion. Walking and turning becomes difficult and stiff-legged. This is soon followed by spasm of neck and back muscles, which can lead to severe extension of neck and an arched back posture known as opisthotonus (not explicitly in notes but a common veterinary term for "extension of neck and back"). The sustained muscle contractions eventually involve the respiratory muscles, leading to laboured breathing (dyspnea), which can culminate in respiratory failure. Ultimately, untreated or severe cases often result in death within 72 hrs after onset of clinical signs, primarily due to respiratory paralysis or exhaustion.

Diagnosis

A tentative diagnosis of tetanus is primarily based on clinical signs as well as history of recent trauma or a wound. The classic signs of trismus, third eyelid prolapse, a "sawhorse" stance, and generalized muscle rigidity are highly suggestive.

Confirmatory diagnosis in a clinical setting can be challenging. While the notes mention demonstrating presence of tetanus toxins from serum, this is often not practical or rapid enough for routine diagnosis in an acute case. Bacterial culture from the wound is also frequently unsuccessful due to the fastidious nature of the organism and its presence in low numbers. Therefore, the definitive diagnosis is largely clinical.

Treatment and control
Treatment of tetanus focuses on neutralizing unbound toxin, eliminating the source of toxin production, and providing intensive supportive care.

1. Wound management:
Open the wound to allow proper aeration, as the causative bacterium (Clostridium tetani) thrives in anaerobic (oxygen-poor) conditions.
2. Cleaning and disinfection:
Clean the wound thoroughly with clean water and disinfect using iodine to reduce bacterial load.
3. Antibiotic therapy:
Administer penicillin either directly at the wound site or intramuscularly (IM) to eliminate the infection.
4. Vaccination:
All at-risk animals should be vaccinated to prevent future infections, especially following trauma or surgical procedure

AMKOVET CONSULT.

Animal never betrayed people.trust them and pertiner them.AMKOVET CONSULT.
24/06/2025

Animal never betrayed people.
trust them and pertiner them.
AMKOVET CONSULT.

Animal Dystocia:******************โฌค ๐˜ฟ๐™”๐™Ž๐™๐™Š๐˜พ๐™„๐˜ผ ๐™„๐™‰ ๐˜ผ๐™‰๐™„๐™ˆ๐˜ผ๐™‡๐™Ž:โœณ๏ธ It is a difficult birth or the inability to expel the fetus t...
20/06/2025

Animal Dystocia:
******************

โฌค ๐˜ฟ๐™”๐™Ž๐™๐™Š๐˜พ๐™„๐˜ผ ๐™„๐™‰ ๐˜ผ๐™‰๐™„๐™ˆ๐˜ผ๐™‡๐™Ž:
โœณ๏ธ It is a difficult birth or the inability to expel the fetus through the birth canal without assistance.

โฌค ๐˜พ๐˜ผ๐™๐™Ž๐™€๐™Ž:
โœ“ Malposition/abnormal presentation.
โœ“ Fetomaternal mismatch
โœ“ Failure of cervical dilation (ringwomb).
โœ“ Vaginal prolapse
โœ“ Uterine torsion/Uterine inertia
โœ“ Pain can also be a factor leading to dystocia.
โœ“ fetal oversize/fetal monsters/diseases/sex

โฌค ๐™ˆ๐˜ผ๐™‰๐˜ผ๐™‚๐™€๐™ˆ๐™€๐™‰๐™ ๐™Š๐™ ๐˜ฟ๐™”๐™Ž๐™๐™Š๐˜พ๐™„๐˜ผ:
โœณ๏ธ If the contractions have stopped, administer oxytocin or calcium gluconate with intravenous (IV) fluids, as well as electrolytes and oxygen.
โœณ๏ธ Result of the abnormal fetal position can be corrected & managed by forceps delivery
โœณ๏ธ Pulling of the fetus via rope attached or direct pull if it's small animal.

โฌค ๐™‹๐™๐™€๐™‘๐™€๐™‰๐™๐™„๐™Š๐™‰:
โœณ๏ธ Good body condition score
โœณ๏ธ Select bulls and cows for calving ease traits.
โœณ๏ธ Pregnancy check cows
โœณ๏ธ Assist in deliveries.

โฌค ๐™€๐™๐™๐™€๐˜พ๐™๐™Ž ๐™Š๐™‰ ๐˜ผ๐™‰๐™„๐™ˆ๐˜ผ๐™‡๐™Ž:
โœณ๏ธ Prolonged hypoxia
significant acidosis
โœณ๏ธ Increased stillborn
โœณ๏ธ Dams trauma, paresis, metritis, endometritis.

AMKOVET CONSULT.

Calcium imbalance.
19/06/2025

Calcium imbalance.

Milk Neck (Milk Goiters) in Goats Milk neck, which may also be called milk goiters, are growths that can form on the nec...
18/06/2025

Milk Neck (Milk Goiters) in Goats
Milk neck, which may also be called milk goiters, are growths that can form on the neck of young goat kids.
Milk goitre is common in some breeds of goat, especially Nubians and Boers. The milk goitre is a soft swelling in the neck on both sides of the larynx, and sometimes under the jaw. The soft swelling is the enlargement of the thymus, but post mortem examination has shown the tissue to be histologically normal.

The swellings begin to appear from one week of age, and on average, reach their largest mass at about four months. After this time spontaneous but slow regression occurs which leaves a flap of loose skin.

Blood samples taken from these animals will show a normal or increased blood iodine, and a normal blood thyroxine level (i.e. this is NOT a thyroid or iodine problem). The thymus gland is a very important part of the immune system, providing 'training' of T-cells to differentiate host cells from foreign invaders. The peak of this activity is in young animals which also have the highest mass of thymus tissue. The large size of the thymus gland should not be considered pathological, and should not be 'treated'.
Any effort to reduce the size of the gland (eg. injecting steroids) will result in a less efficient immunity for that animal.

Do not supplement the goat kid with iodine without consulting with qualified vet. (Iodine toxicity is easy to induce)
- It's just a pocket of loose skin!
AMKOVET CONSULT

Dewormers and uses.๐Ÿš‘๐Ÿš‘๐Ÿš‘๐Ÿ’‰๐Ÿ’‰๐Ÿ’‰๐Ÿ’Š๐Ÿ’Š๐Ÿ’ŠAMKOVET CONSULT.
16/06/2025

Dewormers and uses.
๐Ÿš‘๐Ÿš‘๐Ÿš‘
๐Ÿ’‰๐Ÿ’‰๐Ÿ’‰
๐Ÿ’Š๐Ÿ’Š๐Ÿ’Š
AMKOVET CONSULT.

๐Ÿ“‘ Restraining Techniques in cattle ๐Ÿ„
15/06/2025

๐Ÿ“‘ Restraining Techniques in cattle ๐Ÿ„

Stages of Parturition in BovinesParturition, or the process of giving birth, in bovines occurs in three distinct stages....
14/06/2025

Stages of Parturition in Bovines

Parturition, or the process of giving birth, in bovines occurs in three distinct stages. Understanding these stages is crucial for veterinarians, farmers, and animal health workers to ensure safe delivery and postnatal care.

Stage I: Cervical Dilation (Preparation Stage)

Duration: 2โ€“6 hours (can be longer in heifers)

Signs: Restlessness, tail swishing, lying down and getting up frequently, vocalization, anorexia, and mucus discharge.

Physiology: Uterine contractions begin, and the cervix gradually dilates. The calf repositions for birth.

Stage II: Delivery of the Calf (Expulsion Stage)

Duration: 30 minutes to 2 hours in cows (up to 4 hours in heifers)

Signs: Active straining, visible amniotic sac, followed by the emergence of the calf's feet and head.

Physiology: Strong abdominal and uterine contractions expel the fetus. Assistance is only required if prolonged or if malpresentation occurs.

Stage III: Expulsion of the Placenta (Afterbirth)

Duration: Usually within 6 hours

Signs: Detachment and expulsion of fetal membranes.

Physiology: Uterine contractions continue to help detach and expel the placenta. Retention beyond 12 hours is considered abnormal and requires veterinary attention.

Clinical Tip:
Delayed or prolonged parturition may lead to complications such as dystocia, retained placenta, or uterine infections. Monitoring each stage helps ensure timely intervention and better maternal and calf health.

---

Reference:
Noakes, D. E., Parkinson, T. J., England, G. C. W. (2018). Veterinary Reproduction and Obstetrics (10th ed.). Elsevier.

๐๐ž๐ซ-๐ซ๐ž๐œ๐ญ๐š๐ฅ ๐๐š๐ฅ๐ฉ๐š๐ญ๐ข๐จ๐ง ๐š๐ง๐ ๐„๐ฌ๐ญ๐ซ๐ฎ๐ฌ ๐ƒ๐ž๐ญ๐ž๐œ๐ญ๐ข๐จ๐ง ๐ข๐ง ๐‚๐š๐ญ๐ญ๐ฅ๐ž: ๐Ÿ. ๐๐ž๐ซ-๐ซ๐ž๐œ๐ญ๐š๐ฅ ๐๐š๐ฅ๐ฉ๐š๐ญ๐ข๐จ๐ง๐ƒ๐ž๐Ÿ๐ข๐ง๐ข๐ญ๐ข๐จ๐ง:Per-re**al palpation is a manual ...
11/06/2025

๐๐ž๐ซ-๐ซ๐ž๐œ๐ญ๐š๐ฅ ๐๐š๐ฅ๐ฉ๐š๐ญ๐ข๐จ๐ง ๐š๐ง๐ ๐„๐ฌ๐ญ๐ซ๐ฎ๐ฌ ๐ƒ๐ž๐ญ๐ž๐œ๐ญ๐ข๐จ๐ง ๐ข๐ง ๐‚๐š๐ญ๐ญ๐ฅ๐ž:

๐Ÿ. ๐๐ž๐ซ-๐ซ๐ž๐œ๐ญ๐š๐ฅ ๐๐š๐ฅ๐ฉ๐š๐ญ๐ข๐จ๐ง
๐ƒ๐ž๐Ÿ๐ข๐ง๐ข๐ญ๐ข๐จ๐ง:
Per-re**al palpation is a manual examination technique used to assess the reproductive tract of cattle (and other large animals) by inserting a lubricated arm into the re**um to feel the reproductive organs through the re**al wall.

๐๐ฎ๐ซ๐ฉ๐จ๐ฌ๐ž:
โ€ข Pregnancy diagnosis (as early as 30โ€“35 days).
โ€ข Detection of ovarian structures (corpus luteum, follicles, cysts).
โ€ข Uterine health assessment (size, tone, fluid accumulation).

๐๐ซ๐จ๐œ๐ž๐๐ฎ๐ซ๐ž:
1. Restrain the animal properly to avoid injury.
2. Wear a glove, apply lubricant, and gently insert the hand into the re**um.
3. Locate the cervix as a firm, tubular structure.
4. Trace the uterine horns โ€“ non-pregnant horns are symmetrical; pregnancy causes asymmetry.
5. Check ovaries for follicles (smooth, fluid-filled) or corpus luteum (firm, raised).

๐‚๐ก๐š๐ฅ๐ฅ๐ž๐ง๐ ๐ž๐ฌ:
โ€ข Requires skill to avoid re**al injury.
โ€ข Early pregnancy detection can be tricky (membrane slip technique helps).

๐Ÿ. ๐„๐ฌ๐ญ๐ซ๐ฎ๐ฌ (๐‡๐ž๐š๐ญ) ๐ƒ๐ž๐ญ๐ž๐œ๐ญ๐ข๐จ๐ง ๐“๐ž๐œ๐ก๐ง๐ข๐ช๐ฎ๐ž๐ฌ
Why is it important?
โ€ข Optimal AI (Artificial Insemination) timing (12โ€“18 hours after estrus onset).
โ€ข Improves herd fertility and productivity.

๐Œ๐ž๐ญ๐ก๐จ๐๐ฌ:
1. Visual Signs:
โ€ข Standing to be mounted (most reliable sign).
โ€ข Vulvar swelling, mucus discharge.
โ€ข Restlessness, reduced milk yield.

2. Tail Painting/Chalk:
โ€ข Paint the tail head; rubbing off indicates mounting activity.

3. Pedometers/Activity Monitors:
โ€ข Increased movement during estrus (useful in automated systems).

4. Hormonal Assays:
โ€ข Progesterone testing (low progesterone = estrus).

5. Electronic Heat Detectors (e.g., HeatWatch):
โ€ข Mounting sensors send alerts.

๐‚๐จ๐ฆ๐ฆ๐จ๐ง ๐„๐ซ๐ซ๐จ๐ซ๐ฌ:
โ€ข Missing short estrus periods (average: 8โ€“12 hours).
โ€ข Confusing false heats (due to cysts or stress).

๐‘๐ž๐Ÿ๐ž๐ซ๐ž๐ง๐œ๐ž๐ฌ:
1. Noakes, D. E., Parkinson, T. J., & England, G. C. W. (2018). Veterinary Reproduction and Obstetrics* (10th ed.). Elsevier.

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243 Karkasara Way, Gyadi Gyadi 700101, Kano
Abuja

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